Linked Life Data

892011

Source:http://linkedlifedata.com/resource/pubmed/id/892011

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
10
pubmed:dateCreated
1977-10-20
pubmed:abstractText
It has been suggested that vagal contribution to the inspiratory inhibition can be estimated from the difference between the amplitudes of the 'integrated' phrenic activity with and without phasic vagal feedback. At any given body temperature, there is a linear relationship between estimated vagal contribution to the 'off-switch' mechanism and the 'integrated' vagal activity recorded directly. The temperature displaces this relationship to facilitate vagal central effect. There are evidences that a temperature related activity, which is mediated from hypothalamic thermosensitive structures, may directly influence the vagal input to the R beta neurons of the dorsal group of respiratory neurons in the medulla. An increase in Paco2 results in an increase in the peak 'integrated' vagal activity and in the estimated vagal central effect. However, the inhibitory effect of CO2 on pulmonary stretch receptors seems to be enhanced by an increase in body temperature.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Sep
pubmed:issn
0014-9446
pubmed:author
pubmed:issnType
Print
pubmed:volume
36
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2395-9
pubmed:dateRevised
2003-11-14
pubmed:meshHeading
pubmed:year
1977
pubmed:articleTitle
Vagal contribution to the inspiratory 'off-switch' mechanism.
pubmed:publicationType
Journal Article