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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
2
|
| pubmed:dateCreated |
1996-11-20
|
| pubmed:abstractText |
Ataxia telangiectasia (AT) is an autosomal recessive disorder associated with radiation sensitivity and an increased incidence of leukemia, lymphoma, and some solid tumors. After exposure to ionizing radiation, cells from patients with AT demonstrate an attenuated G1-phase checkpoint. Because c-jun is known to regulate, in part, the exit from G1 and the onset of DNA replication, we analyzed c-jun transcription in irradiated AT fibroblasts.
|
| pubmed:grant | |
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Sep
|
| pubmed:issn |
0360-3016
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:day |
1
|
| pubmed:volume |
36
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
355-60
|
| pubmed:dateRevised |
2007-11-14
|
| pubmed:meshHeading |
pubmed-meshheading:8892460-Ataxia Telangiectasia,
pubmed-meshheading:8892460-Chloramphenicol O-Acetyltransferase,
pubmed-meshheading:8892460-Fibroblasts,
pubmed-meshheading:8892460-G1 Phase,
pubmed-meshheading:8892460-Gene Expression,
pubmed-meshheading:8892460-Genes, jun,
pubmed-meshheading:8892460-Humans,
pubmed-meshheading:8892460-Transcription, Genetic,
pubmed-meshheading:8892460-Transfection
|
| pubmed:year |
1996
|
| pubmed:articleTitle |
Prolonged c-jun expression in irradiated ataxia telangiectasia fibroblasts.
|
| pubmed:affiliation |
Department of Radiation and Cellular Oncology, University of Chicago, IL 60637, USA.
|
| pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
|