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| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C0006837 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C0017861 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C0752312 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C0450254 | lld:lifeskim |
| pubmed-article:8824643 | lifeskim:mentions | umls-concept:C1334043 | lld:lifeskim |
| pubmed-article:8824643 | pubmed:issue | 19 | lld:pubmed |
| pubmed-article:8824643 | pubmed:dateCreated | 1996-11-26 | lld:pubmed |
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| pubmed-article:8824643 | pubmed:abstractText | The Candida albicans HOG1 gene (HOG1CA) was cloned by functional complementation of the osmosensitive phenotype associated with Saccharomyces cerevisiae hog1 delta mutants. HOG1CA codes for a 377-amino-acid protein, 78% identical to S. cerevisiae Hog1p. A C. albicans hog1 null mutant was found to be sensitive to osmotic stress and failed to accumulate glycerol on high-osmolarity media. | lld:pubmed |
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| pubmed-article:8824643 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8824643 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8824643 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8824643 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:8824643 | pubmed:issn | 0021-9193 | lld:pubmed |
| pubmed-article:8824643 | pubmed:author | pubmed-author:NombelaCC | lld:pubmed |
| pubmed-article:8824643 | pubmed:author | pubmed-author:San JoséCC | lld:pubmed |
| pubmed-article:8824643 | pubmed:author | pubmed-author:PlaJJ | lld:pubmed |
| pubmed-article:8824643 | pubmed:author | pubmed-author:MongeR ARA | lld:pubmed |
| pubmed-article:8824643 | pubmed:author | pubmed-author:Pérez-DíazRR | lld:pubmed |
| pubmed-article:8824643 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8824643 | pubmed:volume | 178 | lld:pubmed |
| pubmed-article:8824643 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8824643 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8824643 | pubmed:pagination | 5850-2 | lld:pubmed |
| pubmed-article:8824643 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:8824643 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8824643 | pubmed:articleTitle | The mitogen-activated protein kinase homolog HOG1 gene controls glycerol accumulation in the pathogenic fungus Candida albicans. | lld:pubmed |
| pubmed-article:8824643 | pubmed:affiliation | Departamento de Microbiología II, Facultad de Farmacia, Universidad Complutense de Madrid, Spain. | lld:pubmed |
| pubmed-article:8824643 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8824643 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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