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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
1996-10-3
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pubmed:abstractText |
Although the mechanism of preretinal neovascular growth in the cell-injected rabbit eye model is not known, it has been proposed that the initial vasodilation and eventual development of neovascularization may be attributable to inflammatory mediators. However, an alternative explanation involving hypoxia has not been considered. The purpose of this study was to measure preretinal oxygen tension prior to the development of preretinal neovascularization in the cell-injected rabbit eye.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0721-832X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
234
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
43-6
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:8750849-Animals,
pubmed-meshheading:8750849-Anoxia,
pubmed-meshheading:8750849-Disease Models, Animal,
pubmed-meshheading:8750849-Female,
pubmed-meshheading:8750849-Fibroblasts,
pubmed-meshheading:8750849-Fluorocarbons,
pubmed-meshheading:8750849-Magnetic Resonance Spectroscopy,
pubmed-meshheading:8750849-Male,
pubmed-meshheading:8750849-Oxygen,
pubmed-meshheading:8750849-Rabbits,
pubmed-meshheading:8750849-Retinal Neovascularization,
pubmed-meshheading:8750849-Skin,
pubmed-meshheading:8750849-Vitreous Body
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pubmed:year |
1996
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pubmed:articleTitle |
Hypoxia precedes the development of experimental preretinal neovascularization.
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pubmed:affiliation |
Duke Eye Center, Durham, North Carolina, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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