Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
9
pubmed:dateCreated
1996-7-22
pubmed:abstractText
More than 50% of breast ductal carcinomas in situ (DCIS) contain significant histologic necrosis, an important prognostic factor for determining recurrence and progression to invasive breast cancer. We have examined whether the mechanism of this spontaneous cell death might be apoptosis, a genetically encoded suicide pathway that may be triggered by various events including dysregulated cell proliferation.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0008-543X
pubmed:author
pubmed:issnType
Print
pubmed:day
1
pubmed:volume
77
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1831-5
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed-meshheading:8646681-Apoptosis, pubmed-meshheading:8646681-Breast Neoplasms, pubmed-meshheading:8646681-Carcinoma, Ductal, Breast, pubmed-meshheading:8646681-Carcinoma in Situ, pubmed-meshheading:8646681-Cell Death, pubmed-meshheading:8646681-Cell Division, pubmed-meshheading:8646681-Cell Nucleus, pubmed-meshheading:8646681-Coloring Agents, pubmed-meshheading:8646681-DNA Damage, pubmed-meshheading:8646681-DNA Nucleotidylexotransferase, pubmed-meshheading:8646681-Deoxyuracil Nucleotides, pubmed-meshheading:8646681-Disease Progression, pubmed-meshheading:8646681-Female, pubmed-meshheading:8646681-Genes, p53, pubmed-meshheading:8646681-Humans, pubmed-meshheading:8646681-Immunohistochemistry, pubmed-meshheading:8646681-Mutation, pubmed-meshheading:8646681-Necrosis, pubmed-meshheading:8646681-Neoplasm Invasiveness, pubmed-meshheading:8646681-Neoplasm Recurrence, Local, pubmed-meshheading:8646681-Prognosis
pubmed:year
1996
pubmed:articleTitle
Extensive apoptosis in ductal carcinoma in situ of the breast.
pubmed:affiliation
Division of Pediatric Oncology, Dana Farber Cancer Institute, Boston, Massachusetts, USA.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't