| pubmed-article:8641001 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0026820 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0450127 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0225897 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0132555 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:8641001 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:8641001 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:8641001 | pubmed:dateCreated | 1996-7-15 | lld:pubmed |
| pubmed-article:8641001 | pubmed:abstractText | The mechanisms underlying cardiac contractile dysfunction after transplantation remain poorly defined. Previous work has revealed that inducible nitric oxide synthase (iNOS) is expressed in the rat heterotopic cardiac allograft during rejection; resultant overproduction of nitric oxide (NO) might cause cardiac contractile dysfunction via the negative inotropic and cytotoxic actions of NO. In this investigation, we tested the hypothesis that induction of iNOS may occur and be associated with cardiac allograft contractile dysfunction in humans. | lld:pubmed |
| pubmed-article:8641001 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8641001 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:8641001 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8641001 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8641001 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:8641001 | pubmed:issn | 0009-7322 | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:HuntS ASA | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:BillinghamM... | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:CookeJ PJP | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:TsaiP YPY | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:LewisN PNP | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:JohnsR ARA | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:von der... | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:XueCC | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:ValantineH... | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:HaywoodG AGA | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:FowlerM BMB | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:RickenbacherP... | lld:pubmed |
| pubmed-article:8641001 | pubmed:author | pubmed-author:TrindadeP TPT | lld:pubmed |
| pubmed-article:8641001 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8641001 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:8641001 | pubmed:volume | 93 | lld:pubmed |
| pubmed-article:8641001 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8641001 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8641001 | pubmed:pagination | 720-9 | lld:pubmed |
| pubmed-article:8641001 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
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| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:meshHeading | pubmed-meshheading:8641001-... | lld:pubmed |
| pubmed-article:8641001 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8641001 | pubmed:articleTitle | Induction of nitric oxide synthase in the human cardiac allograft is associated with contractile dysfunction of the left ventricle. | lld:pubmed |
| pubmed-article:8641001 | pubmed:affiliation | Division of Cardiovascular Medicine, Stanford (Calif) University School of Medicine, USA. nlewis@virginia.edu | lld:pubmed |
| pubmed-article:8641001 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8641001 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8641001 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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