| pubmed-article:8624510 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C0162740 | lld:lifeskim |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C0038859 | lld:lifeskim |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C1324497 | lld:lifeskim |
| pubmed-article:8624510 | lifeskim:mentions | umls-concept:C1527180 | lld:lifeskim |
| pubmed-article:8624510 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:8624510 | pubmed:dateCreated | 1996-6-27 | lld:pubmed |
| pubmed-article:8624510 | pubmed:abstractText | By screening suppressor mutants of the hy2 mutation of Arabidopsis thaliana, two dominant photomorphogenic mutants, shy1-1D and shy2-1D, for two genetic loci designated as SHY1 and SHY2 (suppressor of hy2 mutation) have been isolated. Both of these non-allelic, extragenic suppressor mutations of hy2 are located on chromosome 1 of the Arabidopsis genome. Both mutations suppress the elongated hypocotyl phenotype of hy2 by light-independent inhibition of hypocotyl growth as well as by increasing the effectiveness of light inhibition of hypocotyl elongation. The shy1-1D mutation is partially photomorphogenic in darkness with apical hook opening and reduced hypocotyl elongation. The shy2-1D mutant displays highly photomorphogenic characteristics in darkness such as true leaf development, cotyledon expansion and extremely reduced hypocotyl growth. In regard to hypocotyl elongation, however, the shy2-1D mutation is still light sensitive. Examination of red-far-red light responses shows that the shy1-1D mutation suppresses the hypocotyl elongation of the hy2 mutation effectively in red light but not effectively in far-red light. The shy2-1D suppresses hypocotyl elongation of the hy2 mutation effectively in both red and far-red light. Both mutations can also suppress the early-flowering phenotype of hy2 and have a distinct pleiotropic effect on leaf development such as upward leaf rolling. The data obtained suggest that SHY1 and SHY2 represent a novel class of components involved in the photomorphogenic pathways of Arabidopsis. This is the first report on the identification of dominant mutations in the light signal transduction pathway of plants. | lld:pubmed |
| pubmed-article:8624510 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8624510 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8624510 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8624510 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8624510 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:8624510 | pubmed:issn | 0960-7412 | lld:pubmed |
| pubmed-article:8624510 | pubmed:author | pubmed-author:FuruyaMM | lld:pubmed |
| pubmed-article:8624510 | pubmed:author | pubmed-author:HwaT STS | lld:pubmed |
| pubmed-article:8624510 | pubmed:author | pubmed-author:KangB JBJ | lld:pubmed |
| pubmed-article:8624510 | pubmed:author | pubmed-author:KimB CBC | lld:pubmed |
| pubmed-article:8624510 | pubmed:author | pubmed-author:SonM AMA | lld:pubmed |
| pubmed-article:8624510 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8624510 | pubmed:volume | 9 | lld:pubmed |
| pubmed-article:8624510 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8624510 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8624510 | pubmed:pagination | 441-56 | lld:pubmed |
| pubmed-article:8624510 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:8624510 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8624510 | pubmed:articleTitle | Two dominant photomorphogenic mutations of Arabidopsis thaliana identified as suppressor mutations of hy2. | lld:pubmed |
| pubmed-article:8624510 | pubmed:affiliation | Department of Life Science, Pohang University of Science and Technology, Kyungbuk, South Korea. | lld:pubmed |
| pubmed-article:8624510 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8624510 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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