| pubmed-article:8610438 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0042776 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0042216 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0042769 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0449297 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0205224 | lld:lifeskim |
| pubmed-article:8610438 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
| pubmed-article:8610438 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:8610438 | pubmed:dateCreated | 1996-5-30 | lld:pubmed |
| pubmed-article:8610438 | pubmed:abstractText | Recent reports have highlighted a potential antiviral activity for nitric oxide (NO). The purpose of this study was to investigate the production of NO in mice during vaccinia virus (VV) or herpes simplex virus type 1 infection, and to assess the role of NO in clearance of VV. Reactive nitrogen intermediates (RNI; NO and its stable oxidation products, nitrite and nitrate) were significantly elevated in the plasma of mice infected with these viruses. Furthermore, spleen cells from virus-infected mice produced elevated RNI levels following stimulation in vitro with LPS. NO production during VV infection was critically dependent on the cytokines tumor necrosis factor and interferon-gamma, and on the presence of both CD4+ and CD8+ T lymphocytes. Treatment of VV-infected mice with the nitric oxide synthase inhibitor N(G)-methyl-L-arginine did not alter the course of infection, suggesting that NO may not be essential for the clearance of this virus. | lld:pubmed |
| pubmed-article:8610438 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8610438 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8610438 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8610438 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8610438 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:8610438 | pubmed:issn | 0042-6822 | lld:pubmed |
| pubmed-article:8610438 | pubmed:author | pubmed-author:RamshawI AIA | lld:pubmed |
| pubmed-article:8610438 | pubmed:author | pubmed-author:CowdenW BWB | lld:pubmed |
| pubmed-article:8610438 | pubmed:author | pubmed-author:RockettK AKA | lld:pubmed |
| pubmed-article:8610438 | pubmed:author | pubmed-author:RubyJJ | lld:pubmed |
| pubmed-article:8610438 | pubmed:author | pubmed-author:RolphM SMS | lld:pubmed |
| pubmed-article:8610438 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8610438 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:8610438 | pubmed:volume | 217 | lld:pubmed |
| pubmed-article:8610438 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8610438 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8610438 | pubmed:pagination | 470-7 | lld:pubmed |
| pubmed-article:8610438 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:8610438 | pubmed:meshHeading | pubmed-meshheading:8610438-... | lld:pubmed |
| pubmed-article:8610438 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8610438 | pubmed:articleTitle | Nitric oxide production is increased during murine vaccinia virus infection, but may not be essential for virus clearance. | lld:pubmed |
| pubmed-article:8610438 | pubmed:affiliation | Division of Cell Biology, John Curtin School of Medical Research, Canberra, Australia. | lld:pubmed |
| pubmed-article:8610438 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8610438 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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