Linked Life Data

8608459

Source:http://linkedlifedata.com/resource/pubmed/id/8608459

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
1996-5-30
pubmed:abstractText
Drugs that prolong cardiac refractoriness can decrease defibrillation energy requirements. In particular, barium, a relatively selective blocker of cardiac Ik1 channels, produces marked decreases in defibrillation energy. The mechanism of this effect is unknown, and may relate to modulation of the effect of defibrillatory shocks, or an alteration of the pattern of ventricular fibrillation (VF) by the drug.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
0828-282X
pubmed:author
pubmed:issnType
Print
pubmed:volume
12
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
399-406
pubmed:dateRevised
2008-4-9
pubmed:meshHeading
pubmed:year
1996
pubmed:articleTitle
Order in disorder: effect of barium on ventricular fibrillation.
pubmed:affiliation
Department of Medicine, University of Toronto, Ontario. dorianp@smh.toronto.on.ca
pubmed:publicationType
Journal Article