Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
10
pubmed:dateCreated
1996-3-7
pubmed:abstractText
A population-based study of active epilepsy in mentally retarded children identified 98 children, 6-13 years old. A biopathological origin was established in 66% of mildly and 92% of severely retarded children: a prenatal etiology was considered in 51% and 57%, a perinatal in 9% and 19%, a postnatal in 6% and 16% and an untraceable etiology in 34% and 8%, respectively. Severe mental retardation was more frequent in the peri- and postnatal groups (80% and 83%) than in the prenatal and untraceable groups (67% and 29%). Thirty-four pre- and perinatal optimal items were defined. Children with a prenatal etiology did not differ from controls in any of the periods. Children with a perinatal etiology had, compared with controls, higher proportions of non-optimal items successively increasing through the pre- and perinatal periods showing the accumulation of negative events.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
0803-5253
pubmed:author
pubmed:issnType
Print
pubmed:volume
84
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1153-9
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed:year
1995
pubmed:articleTitle
Active epilepsy in mentally retarded children. II. Etiology and reduced pre- and perinatal optimality.
pubmed:affiliation
Department of Pediatrics, University of Göteborg, Ostra Hospital, Sweden.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't