rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
5
|
pubmed:dateCreated |
1996-2-20
|
pubmed:abstractText |
The underlying mechanisms involved in the pathogenesis of amenorrhoea in hyperprolactinaemic states still remain unclear. Conflicting information exists on the role of endogenous opiates on gonadotrophin disturbances in this pathological condition. In this study we have undertaken a detailed investigation of LH and PRL secretion before and during administration of naloxone, an opioid receptor blocker, in hyperprolactinaemic women with or without ovarian function in order to assess the role of ovarian steroids upon naloxone induced LH and PRL release.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Nov
|
pubmed:issn |
0300-0664
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
43
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
591-600
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:8548944-Adolescent,
pubmed-meshheading:8548944-Adult,
pubmed-meshheading:8548944-Case-Control Studies,
pubmed-meshheading:8548944-Chromatography, Gel,
pubmed-meshheading:8548944-Female,
pubmed-meshheading:8548944-Gonadotropin-Releasing Hormone,
pubmed-meshheading:8548944-Humans,
pubmed-meshheading:8548944-Hyperprolactinemia,
pubmed-meshheading:8548944-Luteal Phase,
pubmed-meshheading:8548944-Luteinizing Hormone,
pubmed-meshheading:8548944-Naloxone,
pubmed-meshheading:8548944-Narcotic Antagonists,
pubmed-meshheading:8548944-Ovulation,
pubmed-meshheading:8548944-Pituitary Gland,
pubmed-meshheading:8548944-Prolactin
|
pubmed:year |
1995
|
pubmed:articleTitle |
Evidence for an altered luteinizing hormone sensitivity to naloxone in pathological hyperprolactinaemia.
|
pubmed:affiliation |
Department of Reproductive Biology, Instituto Nacional de la Nutrición Salvador Zubirán, Mexico City, Mexico.
|
pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
|