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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
1994-3-17
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pubmed:abstractText |
We hypothesized that endothelin-1 (ET-1) is an important mediator in renal dysfunction under septic conditions. This study clarified the pathophysiologic role of ET-1 in renal function under conditions of surgical stress, especially sepsis.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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|
pubmed:issn |
0039-6060
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
115
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
199-204
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pubmed:dateRevised |
2010-3-24
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pubmed:meshHeading |
pubmed-meshheading:8310408-Animals,
pubmed-meshheading:8310408-Antibodies,
pubmed-meshheading:8310408-Blood Urea Nitrogen,
pubmed-meshheading:8310408-Creatinine,
pubmed-meshheading:8310408-Endothelins,
pubmed-meshheading:8310408-Endotoxins,
pubmed-meshheading:8310408-Hemodynamics,
pubmed-meshheading:8310408-Kidney,
pubmed-meshheading:8310408-Lipopolysaccharides,
pubmed-meshheading:8310408-Male,
pubmed-meshheading:8310408-Rats,
pubmed-meshheading:8310408-Rats, Wistar,
pubmed-meshheading:8310408-Shock, Septic
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pubmed:year |
1994
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|
pubmed:articleTitle |
Pathophysiologic role of endothelin-1 in renal function in rats with endotoxin shock.
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pubmed:affiliation |
Department of Surgery, Keio University School of Medicine, Tokyo, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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