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rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
2
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|
pubmed:dateCreated |
1994-3-10
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|
pubmed:abstractText |
Increased production of nitric oxide has been implicated as a mediator during septic shock and sepsis syndrome. Inhibition of nitric oxide production could be beneficial during endotoxemia to improve the individual's hemodynamic status and possibly outcome.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Feb
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|
pubmed:issn |
0004-0010
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pubmed:author |
|
|
pubmed:issnType |
Print
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pubmed:volume |
129
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pubmed:owner |
NLM
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|
pubmed:authorsComplete |
Y
|
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pubmed:pagination |
142-7; discussion 147-8
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|
pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:8304826-Animals,
pubmed-meshheading:8304826-Arginine,
pubmed-meshheading:8304826-Endotoxins,
pubmed-meshheading:8304826-Escherichia coli,
pubmed-meshheading:8304826-Female,
pubmed-meshheading:8304826-Lipopolysaccharides,
pubmed-meshheading:8304826-Lung,
pubmed-meshheading:8304826-Macrophages,
pubmed-meshheading:8304826-Macrophages, Peritoneal,
pubmed-meshheading:8304826-Mice,
pubmed-meshheading:8304826-NG-Nitroarginine Methyl Ester,
pubmed-meshheading:8304826-Nitric Oxide,
pubmed-meshheading:8304826-Nitrites,
pubmed-meshheading:8304826-Peroxidase,
pubmed-meshheading:8304826-Superoxides,
pubmed-meshheading:8304826-Survival Rate
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pubmed:year |
1994
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pubmed:articleTitle |
Inhibition of nitric oxide synthesis is detrimental during endotoxemia.
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pubmed:affiliation |
Division of Surgical Oncology, University of Pennsylvania School of Medicine, Philadelphia.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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