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pubmed-article:8205689pubmed:abstractTextThe abnormal transmembrane action potentials of subendocardial Purkinje fibers that survive 24 to 48 hours after coronary artery occlusion can be a source of the multiform ventricular tachycardias that occur during this time. A change in the density or function of either or both the T-type and L-type cardiac Ca2+ channels may contribute to the altered electrical activity of these Purkinje myocytes.lld:pubmed
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pubmed-article:8205689pubmed:articleTitleReduced calcium currents in subendocardial Purkinje myocytes that survive in the 24- and 48-hour infarcted heart.lld:pubmed
pubmed-article:8205689pubmed:affiliationDepartment of Pharmacology, College of Physicians and Surgeons, Columbia University, New York, NY 10032.lld:pubmed
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