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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
|
pubmed:dateCreated |
1994-7-14
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pubmed:abstractText |
The abnormal transmembrane action potentials of subendocardial Purkinje fibers that survive 24 to 48 hours after coronary artery occlusion can be a source of the multiform ventricular tachycardias that occur during this time. A change in the density or function of either or both the T-type and L-type cardiac Ca2+ channels may contribute to the altered electrical activity of these Purkinje myocytes.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
0009-7322
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
89
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
|
pubmed:pagination |
2747-59
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:8205689-Animals,
pubmed-meshheading:8205689-Calcium Channels,
pubmed-meshheading:8205689-Cell Survival,
pubmed-meshheading:8205689-Dogs,
pubmed-meshheading:8205689-Male,
pubmed-meshheading:8205689-Myocardial Infarction,
pubmed-meshheading:8205689-Purkinje Cells,
pubmed-meshheading:8205689-Time Factors
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pubmed:year |
1994
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pubmed:articleTitle |
Reduced calcium currents in subendocardial Purkinje myocytes that survive in the 24- and 48-hour infarcted heart.
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pubmed:affiliation |
Department of Pharmacology, College of Physicians and Surgeons, Columbia University, New York, NY 10032.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, U.S. Gov't, P.H.S.
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