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pubmed-article:8160233pubmed:abstractTextStudies showing efficacy with free radical scavengers have been conflicting, and when protection was demonstrated it was attributed to action at the level of the vascular endothelium. The purpose of this study was to test the hypotheses that neuronal free radical formation plays a role in the ischemic cascade and occurs intracellularly and that free radical scavengers, if taken up intracellularly, will protect against hypoxic damage.lld:pubmed
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pubmed-article:8160233pubmed:articleTitleSuperoxide dismutase ameliorates neuronal death from hypoxia in culture.lld:pubmed
pubmed-article:8160233pubmed:affiliationDepartment of Neurology, Albert Einstein College of Medicine, Bronx, NY 10461.lld:pubmed
pubmed-article:8160233pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:8160233pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed