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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
1994-5-13
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pubmed:abstractText |
Studies showing efficacy with free radical scavengers have been conflicting, and when protection was demonstrated it was attributed to action at the level of the vascular endothelium. The purpose of this study was to test the hypotheses that neuronal free radical formation plays a role in the ischemic cascade and occurs intracellularly and that free radical scavengers, if taken up intracellularly, will protect against hypoxic damage.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0039-2499
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
857-62; discussion 862-3
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:8160233-Animals,
pubmed-meshheading:8160233-Brain Ischemia,
pubmed-meshheading:8160233-Cell Death,
pubmed-meshheading:8160233-Cell Hypoxia,
pubmed-meshheading:8160233-Free Radicals,
pubmed-meshheading:8160233-Neurons,
pubmed-meshheading:8160233-Rats,
pubmed-meshheading:8160233-Rats, Sprague-Dawley,
pubmed-meshheading:8160233-Superior Cervical Ganglion,
pubmed-meshheading:8160233-Superoxide Dismutase
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pubmed:year |
1994
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pubmed:articleTitle |
Superoxide dismutase ameliorates neuronal death from hypoxia in culture.
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pubmed:affiliation |
Department of Neurology, Albert Einstein College of Medicine, Bronx, NY 10461.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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