| pubmed-article:8092270 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C0013604 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C0042380 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C1522318 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C1882687 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C0080310 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:8092270 | lifeskim:mentions | umls-concept:C0205178 | lld:lifeskim |
| pubmed-article:8092270 | pubmed:issue | 3 Pt 2 | lld:pubmed |
| pubmed-article:8092270 | pubmed:dateCreated | 1994-10-19 | lld:pubmed |
| pubmed-article:8092270 | pubmed:abstractText | The impact of acute myocardial edema on coronary flow and left ventricular performance was studied in isolated isovolumic rat hearts. After 15 min of aortic perfusion with Krebs-Henseleit buffer, hearts (10/group) were either removed for determination of water content or perfused for another 90 min. Three groups were perfused at a constant pressure of 60, 100, or 140 mmHg, and two groups were perfused at 60 or 140 mmHg with adenosine added. Compared with the 15-min group, there was a significant increase in water content in all groups except the 60-mmHg group (P < 0.005). There was a direct linear relationship between increases in coronary vascular resistance over time and water content (P < 0.0001). A decrease in developed pressure and peak +dP/dt was observed only in those groups that accumulated water. An inverse linear relationship was found between changes in developed pressure and water content (P = 0.0001). Water content had no effect on end-diastolic pressure below 5 ml/g; above 5 ml/g, a direct linear relationship was evident (P = 0.009). The results suggest that myocardial edema increases vascular resistance and decreases systolic performance. End-diastolic pressure is less influenced by edema than either systolic or coronary vascular function. | lld:pubmed |
| pubmed-article:8092270 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8092270 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8092270 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8092270 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8092270 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8092270 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8092270 | pubmed:issn | 0002-9513 | lld:pubmed |
| pubmed-article:8092270 | pubmed:author | pubmed-author:EulerD EDE | lld:pubmed |
| pubmed-article:8092270 | pubmed:author | pubmed-author:SobotkaP APA | lld:pubmed |
| pubmed-article:8092270 | pubmed:author | pubmed-author:RubboliAA | lld:pubmed |
| pubmed-article:8092270 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8092270 | pubmed:volume | 267 | lld:pubmed |
| pubmed-article:8092270 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8092270 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8092270 | pubmed:pagination | H1054-61 | lld:pubmed |
| pubmed-article:8092270 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:8092270 | pubmed:meshHeading | pubmed-meshheading:8092270-... | lld:pubmed |
| pubmed-article:8092270 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:8092270 | pubmed:articleTitle | Effect of acute edema on left ventricular function and coronary vascular resistance in the isolated rat heart. | lld:pubmed |
| pubmed-article:8092270 | pubmed:affiliation | Department of Medicine, Loyola University Medical Center, Maywood, Illinois 60153. | lld:pubmed |
| pubmed-article:8092270 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8092270 | pubmed:publicationType | In Vitro | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:8092270 | lld:pubmed |
