rdf:type |
|
lifeskim:mentions |
umls-concept:C0005821,
umls-concept:C0019054,
umls-concept:C0019145,
umls-concept:C0032914,
umls-concept:C0162739,
umls-concept:C0205251,
umls-concept:C0232338,
umls-concept:C0235996,
umls-concept:C0332197,
umls-concept:C0683598,
umls-concept:C1627358,
umls-concept:C2349975
|
pubmed:issue |
2
|
pubmed:dateCreated |
1994-9-15
|
pubmed:abstractText |
Our purpose was to test the hypothesis that the HELLP (hemolysis, elevated liver enzymes, and low platelets) syndrome is the result of excessive vasoconstriction of the hepatic arterial circulation.
|
pubmed:commentsCorrections |
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:status |
MEDLINE
|
pubmed:month |
Aug
|
pubmed:issn |
0002-9378
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
171
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
526-30
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
|
pubmed:year |
1994
|
pubmed:articleTitle |
Enhancement of hepatic artery resistance to blood flow in preeclampsia in presence or absence of HELLP syndrome (hemolysis, elevated liver enzymes, and low platelets)
|
pubmed:affiliation |
Department of Obstetrics and Gynaecology, University Hospital, Groningen, The Netherlands.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|