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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions |
umls-concept:C0007452,
umls-concept:C0014912,
umls-concept:C0023607,
umls-concept:C0025223,
umls-concept:C0030685,
umls-concept:C0033308,
umls-concept:C0086045,
umls-concept:C0391871,
umls-concept:C0392744,
umls-concept:C0439682,
umls-concept:C0680255,
umls-concept:C0699733,
umls-concept:C0950151,
umls-concept:C1264691,
umls-concept:C1280500,
umls-concept:C1283071,
umls-concept:C1310628,
umls-concept:C1527148,
umls-concept:C1963578
|
| pubmed:issue |
5
|
| pubmed:dateCreated |
1994-9-15
|
| pubmed:abstractText |
Two trials were conducted to determine whether 7-d progestogen treatment beginning on d 17 of the estrous cycle altered 1) ovarian follicular development, 2) serum concentration of estradiol-17 beta (E2) and progesterone (P4), and 3) patterns of release of luteinizing hormone (LH). In both trials, Angus, Angus x Holstein, or Holstein cows 2 to 6 yr of age were randomly assigned to receive either melengestrol acetate (MGA, .5 mg.animal-1.d-1; n = 23), a progesterone-releaseing intravaginal device (PRID, n = 26) or to serve as untreated Controls (n = 14). Ultrasonography and blood sampling were performed daily throughout the experiment beginning on d 3 (Trial 1) or d 9 (Trial 2) of the estrous cycle. In Trial 2, blood samples were collected every 15-min for 6 h on d 17 (all cows) and d 20 and 23 (MGA and PRID cows) for determination of LH. Estrous cycle length was 3 to 5 d greater (P < .05) for MGA- and PRID-treated cows characterized by two (MGA-2 and PRID-2) or three (MGA-3 and PRID-3) dominant follicles than for control cows exhibiting two (Control-2) or three (Control-3) dominant follicles. A greater proportion (P < .05) of MGA- than of PRID-treated cows ovulated the follicle that was dominant at the beginning of treatment on d 17 (10 of 23 vs 1 of 26). Serum P4 concentrations declined 3 d earlier in Control-2 and MGA-2 cows than in Control-3, MGA-3 or PRID-3 cows.(ABSTRACT TRUNCATED AT 250 WORDS)
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
May
|
| pubmed:issn |
0021-8812
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
72
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
1282-9
|
| pubmed:dateRevised |
2003-11-14
|
| pubmed:meshHeading |
pubmed-meshheading:8056675-Administration, Intravaginal,
pubmed-meshheading:8056675-Administration, Oral,
pubmed-meshheading:8056675-Animals,
pubmed-meshheading:8056675-Cattle,
pubmed-meshheading:8056675-Estradiol,
pubmed-meshheading:8056675-Estrus,
pubmed-meshheading:8056675-Female,
pubmed-meshheading:8056675-Least-Squares Analysis,
pubmed-meshheading:8056675-Luteinizing Hormone,
pubmed-meshheading:8056675-Melengestrol Acetate,
pubmed-meshheading:8056675-Ovarian Follicle,
pubmed-meshheading:8056675-Progesterone
|
| pubmed:year |
1994
|
| pubmed:articleTitle |
Effect of melengestrol acetate (MGA) or progesterone-releasing intravaginal device (PRID) on follicular development, concentrations of estradiol-17 beta and progesterone, and luteinizing hormone release during an artificially lengthened bovine estrous cycle.
|
| pubmed:affiliation |
Department of Animal and Poultry Sciences, Virginia Polytechnic Institute & State University, Blacksburg 24061.
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| pubmed:publicationType |
Journal Article
|