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rdf:type |
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|
lifeskim:mentions |
|
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pubmed:issue |
1
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pubmed:dateCreated |
1994-8-2
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pubmed:abstractText |
Septic shock is characterized by systemic vasodilation and an impaired reactivity to vasoconstrictor agents. It has been suggested that an excessive release of nitric oxide has a role in this hemodynamic derangement.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0012-3692
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
106
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
250-6
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:8020279-Animals,
pubmed-meshheading:8020279-Arginine,
pubmed-meshheading:8020279-Dose-Response Relationship, Drug,
pubmed-meshheading:8020279-Escherichia coli Infections,
pubmed-meshheading:8020279-Nitric Oxide,
pubmed-meshheading:8020279-Nitroarginine,
pubmed-meshheading:8020279-Norepinephrine,
pubmed-meshheading:8020279-Sheep,
pubmed-meshheading:8020279-Shock, Septic,
pubmed-meshheading:8020279-Vascular Resistance,
pubmed-meshheading:8020279-Vasoconstriction
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pubmed:year |
1994
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|
pubmed:articleTitle |
Inhibition of nitric oxide synthesis improves the vasoconstrictive effect of noradrenaline in sepsis.
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pubmed:affiliation |
Unidad de Cuidados Intensivos, Hospital Ramón y Cajal, Madrid, Spain.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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