pubmed-article:7948404 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0085112 | lld:lifeskim |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0007613 | lld:lifeskim |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0301944 | lld:lifeskim |
pubmed-article:7948404 | lifeskim:mentions | umls-concept:C0205132 | lld:lifeskim |
pubmed-article:7948404 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:7948404 | pubmed:dateCreated | 1994-12-2 | lld:pubmed |
pubmed-article:7948404 | pubmed:abstractText | HTLV-I-transformed human T-cell line, MT-2, has been reported to successfully proliferate in only the SCID mice with depletion of NK cell function. However, MT-2 cells could be engrafted into SCID mice possessing normal NK cell function in this study. MT-2 cells (5-7 x 10(7) cells) were intraperitoneally injected into SCID mice without the deletion of NK cell function. The SCID mice developed tumors in the peritoneal cavities 3 months after the inoculation of MT-2 cells. All tumors reacted to anti-HTLV-I p19 and anti-HLA-DR monoclonal antibodies by immunofluorescence assay and were also positive for the HTLV-I gene by PCR assay. DNA obtained from main organs in group of mice with or without tumors showed a high incidence of positive signals for HTLV-I gene by PCR assay. | lld:pubmed |
pubmed-article:7948404 | pubmed:language | eng | lld:pubmed |
pubmed-article:7948404 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7948404 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:7948404 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7948404 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7948404 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:7948404 | pubmed:month | Jun | lld:pubmed |
pubmed-article:7948404 | pubmed:issn | 0916-7250 | lld:pubmed |
pubmed-article:7948404 | pubmed:author | pubmed-author:NomuraTT | lld:pubmed |
pubmed-article:7948404 | pubmed:author | pubmed-author:IshibashiKK | lld:pubmed |
pubmed-article:7948404 | pubmed:author | pubmed-author:ShinguMM | lld:pubmed |
pubmed-article:7948404 | pubmed:author | pubmed-author:OhsugiTT | lld:pubmed |
pubmed-article:7948404 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:7948404 | pubmed:volume | 56 | lld:pubmed |
pubmed-article:7948404 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:7948404 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:7948404 | pubmed:pagination | 601-3 | lld:pubmed |
pubmed-article:7948404 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:7948404 | pubmed:meshHeading | pubmed-meshheading:7948404-... | lld:pubmed |
pubmed-article:7948404 | pubmed:year | 1994 | lld:pubmed |
pubmed-article:7948404 | pubmed:articleTitle | Engraftment of HTLV-I-transformed human T-cell line into SCID mice with NK cell function. | lld:pubmed |
pubmed-article:7948404 | pubmed:affiliation | Department of Virology, Kurume University School of Medicine, Osaka University Medical School, Japan. | lld:pubmed |
pubmed-article:7948404 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:7948404 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:7948404 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:7948404 | lld:pubmed |