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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
1976-12-30
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pubmed:abstractText |
Liquid-holding treatment in non-nutrient solutions after U.V.-exposure results in an increased resistance of wild-type (RAD) yeast cells to a second U.V.-treatment (repair resistance). U.V.-sensitive (rad) mutants of yeast which show variation in their response to liquid-holding treatment and a second U.V.-dose-range have been classified into two groups. Mutants of Group 1 show increased viability after post-U.V.-liquid-holding treatment and show repair resistance. In contrast, mutants of Group 2 which show reduced viability during post-U.V.-liquid-holding treatment have the same U.V.-sensitivity, both before and after liquid-holding treatment. Genetic analysis of crosses of the rad mutants to wild-type cultures indicate that the phenotype of repair resistance to U.V.-treatment is under genetic control and depends on the presence of alleles of radiation sensitivity genes, which also confer the property of liquid-holding recovery.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0020-7616
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
30
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
13-24
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pubmed:dateRevised |
2000-12-18
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pubmed:meshHeading | |
pubmed:year |
1976
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pubmed:articleTitle |
The genetic control of liquid-holding recovery and U.V.-induced repair resistance in the yeast, Saccharomyces cerevisiae.
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pubmed:publicationType |
Journal Article
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