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rdf:type |
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lifeskim:mentions |
umls-concept:C0010287,
umls-concept:C0014025,
umls-concept:C0018557,
umls-concept:C0018787,
umls-concept:C0018801,
umls-concept:C0027061,
umls-concept:C0087111,
umls-concept:C0205179,
umls-concept:C0337819,
umls-concept:C0442805,
umls-concept:C0443286,
umls-concept:C0597198,
umls-concept:C1442080,
umls-concept:C2347957
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|
pubmed:issue |
6
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|
pubmed:dateCreated |
1995-4-13
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|
pubmed:abstractText |
Converting enzyme inhibitor treatment of congestive heart failure slows progression to failure and reduces mortality rate. It is known whether these benefits are due solely to improved hemodynamics or also to improved myocyte energetics. This study examines the effect of enalapril treatment on both isovolumic contractile performance and its biochemical correlate, flux through the creatine kinase (CK) system, in an animal model of severely failing myocardium.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
|
|
pubmed:issn |
0009-7322
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:day |
15
|
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pubmed:volume |
91
|
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1824-33
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:7882493-Animals,
pubmed-meshheading:7882493-Body Weight,
pubmed-meshheading:7882493-Creatine Kinase,
pubmed-meshheading:7882493-Cricetinae,
pubmed-meshheading:7882493-Enalapril,
pubmed-meshheading:7882493-Energy Metabolism,
pubmed-meshheading:7882493-Heart Failure,
pubmed-meshheading:7882493-Isoenzymes,
pubmed-meshheading:7882493-Kinetics,
pubmed-meshheading:7882493-Magnetic Resonance Spectroscopy,
pubmed-meshheading:7882493-Male,
pubmed-meshheading:7882493-Mesocricetus,
pubmed-meshheading:7882493-Myocardial Contraction,
pubmed-meshheading:7882493-Myocardium,
pubmed-meshheading:7882493-Organ Size
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pubmed:year |
1995
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pubmed:articleTitle |
Enalapril treatment increases cardiac performance and energy reserve via the creatine kinase reaction in myocardium of Syrian myopathic hamsters with advanced heart failure.
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pubmed:affiliation |
Department of Medicine, Brigham and Women's Hospital, Boston, Mass 02115.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
|
