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pubmed-article:7870111pubmed:abstractTextThe clinical picture of lead neuropathy was classically described as a painless progressive motor neuropathy with axonal loss. The literature review fails to demonstrate a consensus on the site of axonal loss. This is an EMG report of a patient who developed a late lead neuropathy after a shotgun injury. A 69-year-old Filipino, healthy, male nondrinker sustained a shotgun injury to his left elbow. Nineteen years later he developed abdominal pain, followed by generalized weakness, distal greater than proximal in the extremities, and impaired pin-prick, proprioception, and two-point discrimination. He became nonambulatory and totally dependent in daily activities. He was lost to follow-up for 2 years until January 1993 when he presented with a blood lead level of 84 micrograms/dL. EMG examination revealed a sensorimotor peripheral polyneuropathy with severe axonal loss. This case demonstrates that axonal loss is the predominant feature in lead neuropathy and the location of pathology is in the peripheral nerves.lld:pubmed
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pubmed-article:7870111pubmed:authorpubmed-author:KingeryW SWSlld:pubmed
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pubmed-article:7870111pubmed:pagination326-9lld:pubmed
pubmed-article:7870111pubmed:dateRevised2004-11-17lld:pubmed
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pubmed-article:7870111pubmed:year1995lld:pubmed
pubmed-article:7870111pubmed:articleTitleAn EMG case report of lead neuropathy 19 years after a shotgun injury.lld:pubmed
pubmed-article:7870111pubmed:affiliationDepartment of Functional Restoration, Stanford University, California.lld:pubmed
pubmed-article:7870111pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:7870111pubmed:publicationTypeCase Reportslld:pubmed