rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
10
|
pubmed:dateCreated |
1995-7-6
|
pubmed:abstractText |
To elucidate the regulation of cardiac gene expression by mechanical stress and to analyse molecular mechanisms associated with the involvement of angiotensin II (Ang II) in the development of cardiac hypertrophy and dysfunction.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:status |
MEDLINE
|
pubmed:month |
Dec
|
pubmed:issn |
0952-1178
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
12
|
pubmed:geneSymbol |
c-fos
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
S153-7
|
pubmed:dateRevised |
2008-11-21
|
pubmed:meshHeading |
pubmed-meshheading:7769485-Animals,
pubmed-meshheading:7769485-Gene Expression,
pubmed-meshheading:7769485-Genes, fos,
pubmed-meshheading:7769485-Humans,
pubmed-meshheading:7769485-Hypertrophy, Left Ventricular,
pubmed-meshheading:7769485-Renin-Angiotensin System,
pubmed-meshheading:7769485-Stress, Physiological,
pubmed-meshheading:7769485-Ventricular Dysfunction, Left
|
pubmed:year |
1994
|
pubmed:articleTitle |
Involvement of the renin-angiotensin system in the development of left ventricular hypertrophy and dysfunction.
|
pubmed:affiliation |
Department of Medicine III, Tokyo University School of Medicine, Japan.
|
pubmed:publicationType |
Journal Article,
Review,
Research Support, Non-U.S. Gov't
|