| pubmed-article:7732381 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7732381 | lifeskim:mentions | umls-concept:C0684336 | lld:lifeskim |
| pubmed-article:7732381 | lifeskim:mentions | umls-concept:C0020962 | lld:lifeskim |
| pubmed-article:7732381 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:7732381 | lifeskim:mentions | umls-concept:C0052441 | lld:lifeskim |
| pubmed-article:7732381 | lifeskim:mentions | umls-concept:C0239946 | lld:lifeskim |
| pubmed-article:7732381 | pubmed:issue | 5211 | lld:pubmed |
| pubmed-article:7732381 | pubmed:dateCreated | 1995-5-30 | lld:pubmed |
| pubmed-article:7732381 | pubmed:abstractText | The aryl hydrocarbon (Ah) receptor (AHR) mediates many carcinogenic and teratogenic effects of environmentally toxic chemicals such as dioxin. An AHR-deficient (Ahr-/-) mouse line was constructed by homologous recombination in embryonic stem cells. Almost half of the mice died shortly after birth, whereas survivors reached maturity and were fertile. The Ahr-/- mice showed decreased accumulation of lymphocytes in the spleen and lymph nodes, but not in the thymus. The livers of Ahr-/- mice were reduced in size by 50 percent and showed bile duct fibrosis Ahr-/- mice were also nonresponsive with regard to dioxin-mediated induction of genes encoding enzymes that catalyze the metabolism of foreign compounds. Thus, the AHR plays an important role in the development of the liver and the immune system. | lld:pubmed |
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| pubmed-article:7732381 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7732381 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7732381 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7732381 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7732381 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7732381 | pubmed:month | May | lld:pubmed |
| pubmed-article:7732381 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:NebertD WDW | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:KimuraSS | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:LeeS SSS | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:WardJ MJM | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:RudikoffSS | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:GonzalezF JFJ | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:Fernandez-Sal... | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:PineasRR | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:HilbertD MDM | lld:pubmed |
| pubmed-article:7732381 | pubmed:author | pubmed-author:McPhailTT | lld:pubmed |
| pubmed-article:7732381 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7732381 | pubmed:day | 5 | lld:pubmed |
| pubmed-article:7732381 | pubmed:volume | 268 | lld:pubmed |
| pubmed-article:7732381 | pubmed:geneSymbol | Ahr | lld:pubmed |
| pubmed-article:7732381 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7732381 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7732381 | pubmed:pagination | 722-6 | lld:pubmed |
| pubmed-article:7732381 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:7732381 | pubmed:meshHeading | pubmed-meshheading:7732381-... | lld:pubmed |
| pubmed-article:7732381 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7732381 | pubmed:articleTitle | Immune system impairment and hepatic fibrosis in mice lacking the dioxin-binding Ah receptor. | lld:pubmed |
| pubmed-article:7732381 | pubmed:affiliation | Laboratory of Molecular Carcinogenesis, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA. | lld:pubmed |
| pubmed-article:7732381 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7732381 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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