Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5225
pubmed:dateCreated
1995-9-11
pubmed:databankReference
pubmed:abstractText
Plants can recognize pathogens through the action of disease resistance (R) genes, which confer resistance to pathogens expressing unique corresponding avirulence (avr) genes. The molecular basis of this gene-for-gene specificity is unknown. The Arabidopsis thaliana RPM1 gene enables dual specificity to pathogens expressing either of two unrelated Pseudomonas syringae avr genes. Despite this function, RPM1 encodes a protein sharing molecular features with recently described single-specificity R genes. Surprisingly, RPM1 is lacking from naturally occurring, disease-susceptible Arabidopsis accessions.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
0036-8075
pubmed:author
pubmed:issnType
Print
pubmed:day
11
pubmed:volume
269
pubmed:geneSymbol
RPM1, avrB, avrRpm1
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
843-6
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:7638602-Amino Acid Sequence, pubmed-meshheading:7638602-Arabidopsis, pubmed-meshheading:7638602-Arabidopsis Proteins, pubmed-meshheading:7638602-Base Sequence, pubmed-meshheading:7638602-Genes, Bacterial, pubmed-meshheading:7638602-Genes, Plant, pubmed-meshheading:7638602-Genetic Complementation Test, pubmed-meshheading:7638602-Molecular Sequence Data, pubmed-meshheading:7638602-Mutation, pubmed-meshheading:7638602-Open Reading Frames, pubmed-meshheading:7638602-Plant Diseases, pubmed-meshheading:7638602-Plant Proteins, pubmed-meshheading:7638602-Plants, Genetically Modified, pubmed-meshheading:7638602-Polymorphism, Restriction Fragment Length, pubmed-meshheading:7638602-Pseudomonas, pubmed-meshheading:7638602-Transformation, Genetic, pubmed-meshheading:7638602-Virulence
pubmed:year
1995
pubmed:articleTitle
Structure of the Arabidopsis RPM1 gene enabling dual specificity disease resistance.
pubmed:affiliation
Max-Delbrück Laboratory, Köln, Germany.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't