Linked Life Data

7590625

Source:http://linkedlifedata.com/resource/pubmed/id/7590625

Statements in which the resource exists.
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pubmed-article:7590625pubmed:abstractTextThis study reports a direct effect of TRH on amylase secretion from isolated rat exocrine pancreatic acinar cells. TRH inhibited carbachol (10(-5) M)-stimulated amylase secretion by a maximum of 24% at a concentration of 10(-11) M (p < 0.05), but did not affect basal amylase release in concentrations from 10(-13) M to 10(-8) M. Ceruletide (3 x 10(-10) M)-stimulated amylase secretion was maximally reduced by 23% at a TRH concentration of 10(-10) M (p < 0.05). Direct stimulation of protein kinase C-mediated secretion by the diacylglycerol analogue 1-oleoyl-2-acetyl-sn-glycerol (OAG) was not altered by TRH. The TRH metabolite cyclo (His-Pro) did not influence basal or stimulated pancreatic secretion in vitro. These findings point to a TRH-mediated modulation of exocrine pancreatic secretion at the receptor site.lld:pubmed
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pubmed-article:7590625pubmed:dateRevised2009-2-19lld:pubmed
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pubmed-article:7590625pubmed:year1995lld:pubmed
pubmed-article:7590625pubmed:articleTitleInhibitory effect of thyrotropin-releasing hormone on enzyme secretion from isolated rat pancreatic acinar cells.lld:pubmed
pubmed-article:7590625pubmed:affiliationRehabilitationsklinik Neresheim, Germany.lld:pubmed
pubmed-article:7590625pubmed:publicationTypeJournal Articlelld:pubmed