Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
10
pubmed:dateCreated
1995-11-2
pubmed:abstractText
Although the signaling pathway involving polyphosphoinositide (poly-PI) hydrolysis and release of inositol 1,4,5-trisphosphate [Ins(1,4,5)P3] is an important mechanism for regulation of neuronal calcium homeostasis, the effect of cerebral ischemia-reperfusion on this calcium signaling pathway is not well understood. Because activity of this pathway is dependent on availability of ATP, this study is aimed at examining the poly-PI signaling pathway and high-energy metabolites in a rat stroke model.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
0039-2499
pubmed:author
pubmed:issnType
Print
pubmed:volume
26
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1893-900
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:7570745-Adenosine Diphosphate, pubmed-meshheading:7570745-Adenosine Monophosphate, pubmed-meshheading:7570745-Adenosine Triphosphate, pubmed-meshheading:7570745-Animals, pubmed-meshheading:7570745-Brain, pubmed-meshheading:7570745-Calcium, pubmed-meshheading:7570745-Carotid Artery, Common, pubmed-meshheading:7570745-Cerebral Arteries, pubmed-meshheading:7570745-Cerebral Cortex, pubmed-meshheading:7570745-Chromatography, High Pressure Liquid, pubmed-meshheading:7570745-Disease Models, Animal, pubmed-meshheading:7570745-Homeostasis, pubmed-meshheading:7570745-Hydrolysis, pubmed-meshheading:7570745-Inositol 1,4,5-Trisphosphate, pubmed-meshheading:7570745-Ischemic Attack, Transient, pubmed-meshheading:7570745-Neurons, pubmed-meshheading:7570745-Phosphatidylinositol Phosphates, pubmed-meshheading:7570745-Rats, pubmed-meshheading:7570745-Reperfusion, pubmed-meshheading:7570745-Signal Transduction, pubmed-meshheading:7570745-Tritium
pubmed:year
1995
pubmed:articleTitle
Inositol trisphosphate, polyphosphoinositide turnover, and high-energy metabolites in focal cerebral ischemia and reperfusion.
pubmed:affiliation
Biochemistry Department, University of Missouri, Columbia 65212, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S.