pubmed-article:7566114 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:7566114 | lifeskim:mentions | umls-concept:C0035143 | lld:lifeskim |
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pubmed-article:7566114 | lifeskim:mentions | umls-concept:C0206588 | lld:lifeskim |
pubmed-article:7566114 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
pubmed-article:7566114 | pubmed:issue | 6548 | lld:pubmed |
pubmed-article:7566114 | pubmed:dateCreated | 1995-11-8 | lld:pubmed |
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pubmed-article:7566114 | pubmed:abstractText | Thyroid-hormone and retinoic-acid receptors exert their regulatory functions by acting as both activators and repressors of gene expression. A nuclear receptor co-repressor (N-CoR) of relative molecular mass 270K has been identified which mediates ligand-independent inhibition of gene transcription by these receptors, suggesting that the molecular mechanisms of repression by thyroid-hormone and retinoic-acid receptors are analogous to the co-repressor-dependent transcriptional inhibitory mechanisms of yeast and Drosophila. | lld:pubmed |
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pubmed-article:7566114 | pubmed:language | eng | lld:pubmed |
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pubmed-article:7566114 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:7566114 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:7566114 | pubmed:month | Oct | lld:pubmed |
pubmed-article:7566114 | pubmed:issn | 0028-0836 | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:RyanAA | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:KameiYY | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:TorchiaJJ | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:GlossBB | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:GlassC KCK | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:SöderströmMM | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:NäärA MAM | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:HeinzelTT | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:KurokawaRR | lld:pubmed |
pubmed-article:7566114 | pubmed:author | pubmed-author:HörleinA JAJ | lld:pubmed |
pubmed-article:7566114 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:7566114 | pubmed:day | 5 | lld:pubmed |
pubmed-article:7566114 | pubmed:volume | 377 | lld:pubmed |
pubmed-article:7566114 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:7566114 | pubmed:authorsComplete | N | lld:pubmed |
pubmed-article:7566114 | pubmed:pagination | 397-404 | lld:pubmed |
pubmed-article:7566114 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:7566114 | pubmed:year | 1995 | lld:pubmed |
pubmed-article:7566114 | pubmed:articleTitle | Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor. | lld:pubmed |
pubmed-article:7566114 | pubmed:affiliation | Howard Hughes Medical Institute, University of California, San Diego, La Jolla 92093-0648, USA. | lld:pubmed |
pubmed-article:7566114 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:7566114 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:7566114 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:7566114 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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