| pubmed-article:7518614 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C0301896 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C0070410 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C1539477 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C0205164 | lld:lifeskim |
| pubmed-article:7518614 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:7518614 | pubmed:issue | 5171 | lld:pubmed |
| pubmed-article:7518614 | pubmed:dateCreated | 1994-8-15 | lld:pubmed |
| pubmed-article:7518614 | pubmed:abstractText | Two molecular mechanisms of T cell-mediated cytotoxicity, one perforin-based, the other Fas-based, have been demonstrated. To determine the extent of their contribution to T cell-mediated cytotoxicity, a range of effector cells from normal control or perforin-deficient mice were tested against a panel of target cells with various levels of Fas expression. All cytotoxicity observed was due to either of these mechanisms, and no third mechanism was detected. Thus, the perforin- and Fas-based mechanisms may account for all T cell-mediated cytotoxicity in short-term in vitro assays. | lld:pubmed |
| pubmed-article:7518614 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7518614 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7518614 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7518614 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7518614 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:7518614 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:NagataSS | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:GolsteinPP | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:HengartnerHH | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:BürkiKK | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:VignauxFF | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:LedermannBB | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:KägiDD | lld:pubmed |
| pubmed-article:7518614 | pubmed:author | pubmed-author:DepraetereVV | lld:pubmed |
| pubmed-article:7518614 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7518614 | pubmed:day | 22 | lld:pubmed |
| pubmed-article:7518614 | pubmed:volume | 265 | lld:pubmed |
| pubmed-article:7518614 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7518614 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7518614 | pubmed:pagination | 528-30 | lld:pubmed |
| pubmed-article:7518614 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:7518614 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:7518614 | pubmed:articleTitle | Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity. | lld:pubmed |
| pubmed-article:7518614 | pubmed:affiliation | Department of Pathology, University of Zürich, Switzerland. | lld:pubmed |
| pubmed-article:7518614 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7518614 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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