| pubmed-article:7502078 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7502078 | lifeskim:mentions | umls-concept:C0237868 | lld:lifeskim |
| pubmed-article:7502078 | lifeskim:mentions | umls-concept:C0288422 | lld:lifeskim |
| pubmed-article:7502078 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:7502078 | lifeskim:mentions | umls-concept:C0036679 | lld:lifeskim |
| pubmed-article:7502078 | pubmed:issue | 5242 | lld:pubmed |
| pubmed-article:7502078 | pubmed:dateCreated | 1996-1-18 | lld:pubmed |
| pubmed-article:7502078 | pubmed:abstractText | Transcriptional silencing at the HMRa locus of Saccharomyces cerevisiae requires the function of the origin recognition complex (ORC), the replication initiator of yeast. Expression of a Drosophila melanogaster Orc2 complementary DNA in the yeast orc2-1 strain, which is defective for replication and silencing, complemented the silencing defect but not the replication defect; this result indicated that the replication and silencing functions of ORC were separable. The orc2-1 mutation mapped to the region of greatest homology between the Drosophila and yeast proteins. The silent state mediated by DmOrc2 was epigenetic; it was propagated during mitotic divisions in a relatively stable way, whereas the nonsilent state was metastable. In contrast, the silent state was erased during meiosis. | lld:pubmed |
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| pubmed-article:7502078 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:7502078 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7502078 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7502078 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:7502078 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:7502078 | pubmed:author | pubmed-author:BotchanM RMR | lld:pubmed |
| pubmed-article:7502078 | pubmed:author | pubmed-author:LalH MHM | lld:pubmed |
| pubmed-article:7502078 | pubmed:author | pubmed-author:RineJJ | lld:pubmed |
| pubmed-article:7502078 | pubmed:author | pubmed-author:GossenMM | lld:pubmed |
| pubmed-article:7502078 | pubmed:author | pubmed-author:Ehrenhofer-Mu... | lld:pubmed |
| pubmed-article:7502078 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7502078 | pubmed:day | 8 | lld:pubmed |
| pubmed-article:7502078 | pubmed:volume | 270 | lld:pubmed |
| pubmed-article:7502078 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7502078 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7502078 | pubmed:pagination | 1671-4 | lld:pubmed |
| pubmed-article:7502078 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:7502078 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7502078 | pubmed:articleTitle | Separation of origin recognition complex functions by cross-species complementation. | lld:pubmed |
| pubmed-article:7502078 | pubmed:affiliation | Department of Molecular and Cell Biology, University of California, Berkeley 94720, USA. | lld:pubmed |
| pubmed-article:7502078 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7502078 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:7502078 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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