| pubmed-article:7338811 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7338811 | lifeskim:mentions | umls-concept:C0006675 | lld:lifeskim |
| pubmed-article:7338811 | lifeskim:mentions | umls-concept:C0027882 | lld:lifeskim |
| pubmed-article:7338811 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
| pubmed-article:7338811 | lifeskim:mentions | umls-concept:C0521116 | lld:lifeskim |
| pubmed-article:7338811 | lifeskim:mentions | umls-concept:C0553251 | lld:lifeskim |
| pubmed-article:7338811 | pubmed:dateCreated | 1982-6-21 | lld:pubmed |
| pubmed-article:7338811 | pubmed:abstractText | 1. A two-electrode voltage clamp method was used to study Ca inward currents in identified Helix aspersa neurones bathed in 25 mM-Ca, Na-free saline with TEA and 4-AP. 2. Inward currents were blocked by CdCl2. In Cd delayed outward currents appeared at +30 mV. When two identical depolarizations were separated by a gap inward current turned off to the same level during the two pulses up to +20 mV; above this potential the records cross over. 3. The turn-off of inward current at potentials up to +20 mV was not affected by 0.2 mM-quinine, which reduced outward currents at more depolarized potentials. Inward currents declined exponentially over the first 100 msec with a time constant around 60 msec at 0 mV. Double-pulse experiments gave the same time course of turn-off. 4. When Ca inward current was reduced by lowering [Ca]o or by partial block by Cd the rate and extent of turn-off was reduced. 5. The inactivation curve (obtained using a double pulse with gap method) was U-shaped. The curve was not significantly changed by addition of quinine (0.2 mM) or by changing test pulse size. 6. Recovery of inward currents after a depolarizing prepulse was a double-exponential process, with time constants of 120 msec and 9.4 sec at 10--11 degrees C. 7. Our results are discussed in terms of possible Ca-dependent Ca inactivation and in terms of the possibility of development of an outward Ca-dependent K current. | lld:pubmed |
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| pubmed-article:7338811 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:7338811 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7338811 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7338811 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:7338811 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:7338811 | pubmed:author | pubmed-author:StandenN BNB | lld:pubmed |
| pubmed-article:7338811 | pubmed:author | pubmed-author:PlantT DTD | lld:pubmed |
| pubmed-article:7338811 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7338811 | pubmed:volume | 321 | lld:pubmed |
| pubmed-article:7338811 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7338811 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7338811 | pubmed:pagination | 273-85 | lld:pubmed |
| pubmed-article:7338811 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:7338811 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:7338811 | pubmed:articleTitle | Calcium current inactivation in identified neurones of Helix aspersa. | lld:pubmed |
| pubmed-article:7338811 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7338811 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:7338811 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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