Switch to
Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5-6
|
pubmed:dateCreated |
1981-10-29
|
pubmed:abstractText |
The following hypothesis is presented: A dysfunction of the hair cell cilia, either in the form of ciliary pathology or as a temporary loss of ciliary stiffness, both of which are documented, ought to lead to a partial decoupling of the involved hair cells from the tectorial membrane. Consequently, 1) energy transmission should be attenuated (= hearing loss), 2) the noise level at the hair cell input should be increased (= tinnitus) and, owing to the concomitant center-clipping of the signal waveform, 3) the input/output function should become steeper (= recruitment) and 4) the formant structure of speech should be largely destroyed (= relatively poor speech discrimination). The above signs and symptoms are characteristic of a number of acute cochlear disorders and would thus find a common explanation.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:issn |
0001-6489
|
pubmed:author | |
pubmed:issnType |
Print
|
pubmed:volume |
91
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
469-79
|
pubmed:dateRevised |
2007-11-15
|
pubmed:meshHeading |
pubmed-meshheading:7270117-Animals,
pubmed-meshheading:7270117-Cats,
pubmed-meshheading:7270117-Hair Cells, Auditory,
pubmed-meshheading:7270117-Hair Cells, Auditory, Inner,
pubmed-meshheading:7270117-Hearing Loss, Sensorineural,
pubmed-meshheading:7270117-Humans,
pubmed-meshheading:7270117-Kanamycin,
pubmed-meshheading:7270117-Neural Conduction,
pubmed-meshheading:7270117-Recruitment, Neurophysiological,
pubmed-meshheading:7270117-Speech Perception,
pubmed-meshheading:7270117-Tectorial Membrane,
pubmed-meshheading:7270117-Tinnitus
|
pubmed:articleTitle |
Stereociliary dysfunction, a case of sensory hearing loss, recruitment, poor speech discrimination and tinnitus.
|
pubmed:publicationType |
Journal Article
|