| pubmed-article:7216466 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C0010572 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C0085862 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C1299583 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C0439536 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C1524063 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C1608386 | lld:lifeskim |
| pubmed-article:7216466 | lifeskim:mentions | umls-concept:C1549571 | lld:lifeskim |
| pubmed-article:7216466 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:7216466 | pubmed:dateCreated | 1981-6-13 | lld:pubmed |
| pubmed-article:7216466 | pubmed:abstractText | A system for measuring chlamydial lipid synthesis was developed with mouse L cells grown in serum-free modified Waymouth 752/l medium in a shaker culture. Host lipid synthesis was reduced approximately 90% when cells were incubated for 24 h in medium containing cycloheximide (2 micrograms/ml). Lipid metabolism was monitored by measuring the incorporation of [3H]isoleucine into the total lipid of normal and infected cells. The results suggested that lipid synthesis of Chlamydia trachomatis lymphogranuloma venereum (LGV-404L) was not inhibited by cycloheximide treatment when the chlamydiae were grown in L cells, whereas host lipid synthesis was inhibited. Chlamydial lipid metabolism began about 6 to 12 h after infection when the noninfectious reticulate body was found and continually increased until the beginning of the appearance of intracellular infectious elementary bodies at 24 to 30 h. | lld:pubmed |
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| pubmed-article:7216466 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:7216466 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7216466 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7216466 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:7216466 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:7216466 | pubmed:author | pubmed-author:JenkinH MHM | lld:pubmed |
| pubmed-article:7216466 | pubmed:author | pubmed-author:RuddC JCJ | lld:pubmed |
| pubmed-article:7216466 | pubmed:author | pubmed-author:AndersonL ELE | lld:pubmed |
| pubmed-article:7216466 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7216466 | pubmed:volume | 31 | lld:pubmed |
| pubmed-article:7216466 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7216466 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7216466 | pubmed:pagination | 668-73 | lld:pubmed |
| pubmed-article:7216466 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:7216466 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:7216466 | pubmed:articleTitle | Use of cycloheximide to study independent lipid metabolism of Chlamydia trachomatis cultivated in mouse L cells grown in serum-free medium. | lld:pubmed |
| pubmed-article:7216466 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7216466 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:7216466 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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