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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
|
pubmed:dateCreated |
1982-12-2
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pubmed:abstractText |
Phenoxybenzamine can selectively eliminate the s-IPSP, in the presence of anti-cholinesterases that enhance s-IPSP and s-EPSP; and the alpha 2-antagonist, yohimbine, can partially but consistently depress s-IPSP selectively. The results provide positive pharmacological support for the monoaminergic nature of the transmitter for s-IPSP in mammalian sympathetic ganglia and argue against suggestions that the s-IPSP is a direct hyperpolarizing response to acetylcholine.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jun
|
pubmed:issn |
0006-8993
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:day |
24
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pubmed:volume |
242
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
345-9
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:7116140-Animals,
pubmed-meshheading:7116140-Electric Conductivity,
pubmed-meshheading:7116140-Electric Stimulation,
pubmed-meshheading:7116140-Evoked Potentials,
pubmed-meshheading:7116140-Ganglia, Sympathetic,
pubmed-meshheading:7116140-Phenoxybenzamine,
pubmed-meshheading:7116140-Rabbits,
pubmed-meshheading:7116140-Tubocurarine,
pubmed-meshheading:7116140-Yohimbine
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pubmed:year |
1982
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pubmed:articleTitle |
Pharmacological properties and monoaminergic mediation of the slow IPSP, in mammalian sympathetic ganglion.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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