| pubmed-article:7064495 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7064495 | lifeskim:mentions | umls-concept:C0037135 | lld:lifeskim |
| pubmed-article:7064495 | lifeskim:mentions | umls-concept:C0341628 | lld:lifeskim |
| pubmed-article:7064495 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:7064495 | lifeskim:mentions | umls-concept:C0032708 | lld:lifeskim |
| pubmed-article:7064495 | lifeskim:mentions | umls-concept:C0917995 | lld:lifeskim |
| pubmed-article:7064495 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:7064495 | pubmed:dateCreated | 1982-5-12 | lld:pubmed |
| pubmed-article:7064495 | pubmed:abstractText | Adult female Wistar rats were fed continuously over a period of 65 days with a diet containing 0.05% hexachlorobenzene (HCB), silymarin (100 mg/kg) or (+)-cyanidanol-3 (150 mg/kg) alone or together with HCB. On day 49 the porphyrins were increased in the cyanidanol-HCB-group in contrast to the silymarin-HCB- and the HCB-group which showed lower levels during the whole experiment. Compared with the other groups--aminolevulinic acid and porphobilinogen were increased in the cyanidanol/HCB-group. Silymarin and cyanidanol had no effect on the formation of experimental porphyria. On the other hand, silymarin is a stronger inducer of the mixed function monooxygenase system than cyanidanol. The type of the cytochrome P-450 induced by the substances could not be identified with the help of different type-specific inhibitors. | lld:pubmed |
| pubmed-article:7064495 | pubmed:language | ger | lld:pubmed |
| pubmed-article:7064495 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7064495 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7064495 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:7064495 | pubmed:issn | 0044-2771 | lld:pubmed |
| pubmed-article:7064495 | pubmed:author | pubmed-author:GoerzGG | lld:pubmed |
| pubmed-article:7064495 | pubmed:author | pubmed-author:ThierWW | lld:pubmed |
| pubmed-article:7064495 | pubmed:author | pubmed-author:BolsenKK | lld:pubmed |
| pubmed-article:7064495 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7064495 | pubmed:volume | 20 | lld:pubmed |
| pubmed-article:7064495 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7064495 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7064495 | pubmed:pagination | 40-6 | lld:pubmed |
| pubmed-article:7064495 | pubmed:dateRevised | 2009-11-11 | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:meshHeading | pubmed-meshheading:7064495-... | lld:pubmed |
| pubmed-article:7064495 | pubmed:year | 1982 | lld:pubmed |
| pubmed-article:7064495 | pubmed:articleTitle | [Influence of silymarin and (+)-cyanidanol-3 on the HCB-induced porphyria in rats (author's transl)]. | lld:pubmed |
| pubmed-article:7064495 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7064495 | pubmed:publicationType | English Abstract | lld:pubmed |
