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pubmed:abstractText |
A patient presented with post-traumatic hypopituitarism, where transient diabetes insipidus, high prolactin levels and prolonged and delayed TSH rise after TRH suggested a hypothalamic lesion. Although there was no gonadotropin response to LHRH as one could have expected, following repeated administration of LHRH, the LH response returned to normal, thus confirming a hypothalamic cause for his hypogonadism.
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