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Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
|
pubmed:dateCreated |
1984-1-7
|
pubmed:abstractText |
Lidoflazine, flunarizine, and R 51 469 antagonized contractions of isolated canine coronary arteries caused by depolarizing solution or 5-hydroxytryptamine, and the further increase in tension caused by hypoxia during such contractions. The 5-HT2-antagonist ketanserin, but not methysergide, antagonized contractions induced by 5-hydroxytryptamine, but not those by high K+; it reduced further increases in tension caused by hypoxia only during contractions caused by 5-hydroxytryptamine. Augmentation by hypoxia of coronary vasoconstriction appears to be a Ca2+-dependent phenomenon.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:month |
Sep
|
pubmed:issn |
0301-4533
|
pubmed:author | |
pubmed:issnType |
Print
|
pubmed:volume |
265
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
172-6
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading | |
pubmed:year |
1983
|
pubmed:articleTitle |
Effect of Ca2+-entry blockade and serotonergic antagonism on contractions of hypoxic isolated canine coronary arteries.
|
pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
|