rdf:type |
|
lifeskim:mentions |
umls-concept:C0003451,
umls-concept:C0019348,
umls-concept:C0020522,
umls-concept:C0020966,
umls-concept:C0026809,
umls-concept:C0039194,
umls-concept:C0871261,
umls-concept:C1283195,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C2911692
|
pubmed:issue |
4
|
pubmed:dateCreated |
1981-5-13
|
pubmed:abstractText |
An adoptive transfer system was used to investigate the H-2 restriction of delayed-type hypersensitivity (DTH) to herpes simplex virus. A successful DTH transfer was achieved when donor and recipient were compatible at the I-A region, with K and D region compatibility unnecessary. However, the rapid clearance of infectious virus from the inoculation site was found only when the donor and recipients were compatible at H-2K (and presumably D) and I-A regions.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Apr
|
pubmed:issn |
0022-1767
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
126
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1260-2
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:6259256-Animals,
pubmed-meshheading:6259256-Antibodies, Viral,
pubmed-meshheading:6259256-Chromosome Mapping,
pubmed-meshheading:6259256-Female,
pubmed-meshheading:6259256-H-2 Antigens,
pubmed-meshheading:6259256-Herpes Simplex,
pubmed-meshheading:6259256-Hypersensitivity, Delayed,
pubmed-meshheading:6259256-Immunity, Cellular,
pubmed-meshheading:6259256-Immunization, Passive,
pubmed-meshheading:6259256-Male,
pubmed-meshheading:6259256-Mice,
pubmed-meshheading:6259256-Mice, Inbred BALB C,
pubmed-meshheading:6259256-Mice, Inbred C57BL,
pubmed-meshheading:6259256-Mice, Inbred CBA,
pubmed-meshheading:6259256-Simplexvirus,
pubmed-meshheading:6259256-T-Lymphocytes
|
pubmed:year |
1981
|
pubmed:articleTitle |
Cell-mediated immunity in herpes simplex virus-infected mice: H-2 mapping of the delayed-type hypersensitivity response and the antiviral T cell response.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|