pubmed-article:6165020 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6165020 | lifeskim:mentions | umls-concept:C0019630 | lld:lifeskim |
pubmed-article:6165020 | lifeskim:mentions | umls-concept:C0086344 | lld:lifeskim |
pubmed-article:6165020 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:6165020 | lifeskim:mentions | umls-concept:C0178654 | lld:lifeskim |
pubmed-article:6165020 | lifeskim:mentions | umls-concept:C0023509 | lld:lifeskim |
pubmed-article:6165020 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:6165020 | pubmed:dateCreated | 1981-7-23 | lld:pubmed |
pubmed-article:6165020 | pubmed:abstractText | By using clones of murine T cells reactive with alloantigens as well as soluble antigens, it has been possible to demonstrate that Ia antigens, Ir gene phenomena (defined as the ability of immune T cells to recognize antigen), and mixed lymphocyte reaction (MLR)-stimulating determinants encoded within the I-A subregion are different manifestations associated with same product. These studies utilized the I-Ab mutant mouse B6.C-H-2bm (bm12), whose defect in the normal expression of the cell surface products of the I-Ab subregion can be partially circumvented through trans-complementation by deriving hybrids between bm12 and mice expressing normal I-A subregion products. Such mice [e.g., (bm12 X B10.A)F1] express several serologically normal I-Ab products but have defects in certain I-A subregion gene products normally expressed on cells of H-2a X H-2b heterozygote mice that are used as restricting elements for antigen recognition by antigen-reactive T cell clones or recognized as alloantigens by alloreactive T cell clones. This defect in cell surface expression of certain normal I-Ab products on the mutant bm12 cells has allowed us to suggest that there exist trans-complementing products on H-2a X H-2b heterozygote mice consisting of Ab alpha Ak beta and Ak alpha Ab beta that restrict antigen recognition by cloned T cells, stimulate MLR responses by cloned T cells, and react with certain anti-Ia antisera. | lld:pubmed |
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pubmed-article:6165020 | pubmed:language | eng | lld:pubmed |
pubmed-article:6165020 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6165020 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:6165020 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6165020 | pubmed:month | Mar | lld:pubmed |
pubmed-article:6165020 | pubmed:issn | 0027-8424 | lld:pubmed |
pubmed-article:6165020 | pubmed:author | pubmed-author:DavidC SCS | lld:pubmed |
pubmed-article:6165020 | pubmed:author | pubmed-author:FathmanC GCG | lld:pubmed |
pubmed-article:6165020 | pubmed:author | pubmed-author:MelvoldRR | lld:pubmed |
pubmed-article:6165020 | pubmed:author | pubmed-author:KimotoMM | lld:pubmed |
pubmed-article:6165020 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6165020 | pubmed:volume | 78 | lld:pubmed |
pubmed-article:6165020 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6165020 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6165020 | pubmed:pagination | 1853-7 | lld:pubmed |
pubmed-article:6165020 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:6165020 | pubmed:year | 1981 | lld:pubmed |
pubmed-article:6165020 | pubmed:articleTitle | Reconstitution of Ir genes, Ia antigens, and mixed lymphocyte reaction determinants by gene complementation. | lld:pubmed |
pubmed-article:6165020 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:6165020 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:6165020 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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