Linked Life Data

6126261

Source:http://linkedlifedata.com/resource/pubmed/id/6126261

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
1982-12-2
pubmed:abstractText
alpha-Bungarotoxin has been proposed to interact with a membrane site in neuronal tissue which has the characteristics of a nicotinic acetylcholine receptor and also a trophic receptor. A nerve cell function which is affected by both nicotinic stimulation and nerve growth factor is the induction of tyrosine hydroxylase. For this reason, alpha-bungarotoxin was tested on the carbachol-induced increase and nerve growth factor-mediated increase in tyrosine hydroxylase activity in two preparations of neuronal origin, organ cultures of rat superior cervical ganglia and cultured bovine adrenal medullary cells. The results demonstrate that the alpha-bungarotoxin site is not involved in tyrosine hydroxylase induction.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0006-8993
pubmed:author
pubmed:issnType
Print
pubmed:day
29
pubmed:volume
244
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
331-6
pubmed:dateRevised
2010-6-4
pubmed:meshHeading
pubmed:year
1982
pubmed:articleTitle
The alpha-bungarotoxin site and its relation to the cholinergic and nerve growth factor mediated increases in tyrosine hydroxylase activity in cultures of sympathetic ganglia and chromaffin cells.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't