Linked Life Data

6089045

Source:http://linkedlifedata.com/resource/pubmed/id/6089045

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
3
pubmed:dateCreated
1984-10-15
pubmed:abstractText
Recent electrophysiological studies on the rat hippocampus (in vivo and in vitro) provide further evidence that neuronal and glial uptake of the inhibitory transmitter gamma-aminobutyric acid (GABA) limits the intensity and the duration of effects not only of locally applied exogenous GABA but also of GABAergic inhibitory synaptic potentials (IPSPs). There is good reason to believe that such uptake is at least partly responsible for the 'fading' of GABA action. Moreover, because it is probably driven by the transmembrane Na+ electrochemical gradient and is accompanied by Na+ influx, GABA uptake is potentially electrogenic and therefore may have a depolarizing effect on both neurons and glia.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jun
pubmed:issn
0304-3940
pubmed:author
pubmed:issnType
Print
pubmed:day
29
pubmed:volume
47
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
283-7
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:year
1984
pubmed:articleTitle
Some functional consequences of GABA uptake by brain cells.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't