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pubmed:abstractText |
Increased survival of density-inhibited Chinese hamster cells after a variety of treatments is analyzed in terms of half-time of "repair," magnitude of repair, and dependence upon nutrient medium during the repair interval. At least three different systems are identified: X-ray-like, UV-like, and an entirely different system after heat shock. Repair after methyl methanesulfonate and after visible light (in bromodeoxyuridine-substituted cells) may be interpreted as a superposition of the X-ray and UV-like repair systems.
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