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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
|
pubmed:dateCreated |
1972-5-16
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pubmed:keyword |
http://linkedlifedata.com/resource/pubmed/keyword/Biology,
http://linkedlifedata.com/resource/pubmed/keyword/Endocrine Effects,
http://linkedlifedata.com/resource/pubmed/keyword/Endocrine System,
http://linkedlifedata.com/resource/pubmed/keyword/Hematological Effects,
http://linkedlifedata.com/resource/pubmed/keyword/Hemic System,
http://linkedlifedata.com/resource/pubmed/keyword/Physiology,
http://linkedlifedata.com/resource/pubmed/keyword/Serum Calcium Level--analysis,
http://linkedlifedata.com/resource/pubmed/keyword/Thyroid Effects
|
pubmed:language |
cze
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:month |
Mar
|
pubmed:issn |
0036-5327
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
74
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pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
81-5
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pubmed:dateRevised |
2009-11-11
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pubmed:otherAbstract |
PIP: The authors describe histologically confirmed medullary carcinoma of the thyroid gland in a living patient. The tumor produces excessive amounts of calcitonin and releases it into the blood stream. In healthy subjects, the plasma concentration of calcitonin is 0.2-1.0 mU MRC/ml plasma; in medullary carcinoma it is 5.66 mU/ml. The tumor responds to artificially induced hypercalcemia by a further increase of plasma calcitonin. 60-120 minutes after a hypercalcemic stimulus, the calcitonin level declines well below the initial value but remains elevated. The patient has a permanently normal blood calcium level. This is probably due to secondary hyperparathyroidism, a compensatory phenomenon. (Author's modified)
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pubmed:meshHeading | |
pubmed:year |
1972
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pubmed:articleTitle |
[Medullary carcinoma of the thyroid gland producing calcitonin].
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pubmed:publicationType |
Journal Article,
English Abstract
|