| pubmed-article:4847244 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C0018802 | lld:lifeskim |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C0348013 | lld:lifeskim |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C0205276 | lld:lifeskim |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:4847244 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:4847244 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:4847244 | pubmed:dateCreated | 1974-10-4 | lld:pubmed |
| pubmed-article:4847244 | pubmed:abstractText | Since the concept of an elevated venous tone in congestive heart failure (CHF) has been recently questioned, the venous volume of the elevated calf at a venous pressure of 30 mm Hg (VV[30]) was determined in 18 normal volunteers (N) and 10 CHF patients with a mercury-in-rubber strain gauge plethysmograph. CHF patients had a significantly lower VV[30] at rest and after intra-arterial phentolamine (2 mg) than normal subjects, suggesting that in these patients a state of peripheral venoconstriction existed (rest-N: 4.63+/-0.17, CHF: 1.7+/-0.23 ml/100 ml, P < 0.01; pre- and postphentolamine-N: 4.85+/-0.21 to 4.95+/-0.31, CHF: 2.26+/-0.29 to 2.68+/-0.38 ml/100 ml, P < 0.01). Of note is that alpha adrenergic blockade failed to increase VV[30] significantly in N, but did increase it in CHF (P < 0.05), suggesting that part of the decreased VV[30] in CHF in due to an augmented sympathoadrenal discharge. When sodium nitrite (30 mg) was given as a single intra-arterial injection before or after phentolamine or when given in four successive doses at 3-min intervals, the VV[30] of CHF patients was never increased to more than 3.62+/-0.42 ml/100 ml and was always less than N (P < 0.01). Importantly, VV[30] in CHF after these interventions was even significantly less than that of N before intervention (P < 0.05), suggesting that factors other than local active smooth muscle venoconstriction were operative in CHF to lower VV[30]. It is suggested that perhaps clinically undetectable edema and an elevated tissue pressure may account for these differences. | lld:pubmed |
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| pubmed-article:4847244 | pubmed:language | eng | lld:pubmed |
| pubmed-article:4847244 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:4847244 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:4847244 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:4847244 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:4847244 | pubmed:issn | 0021-9738 | lld:pubmed |
| pubmed-article:4847244 | pubmed:author | pubmed-author:ZelisRR | lld:pubmed |
| pubmed-article:4847244 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:4847244 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:4847244 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:4847244 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:4847244 | pubmed:pagination | 219-24 | lld:pubmed |
| pubmed-article:4847244 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:4847244 | pubmed:year | 1974 | lld:pubmed |
| pubmed-article:4847244 | pubmed:articleTitle | The contribution of local factors to the elevated venous tone of congestive heart failure. | lld:pubmed |
| pubmed-article:4847244 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:4847244 | pubmed:publicationType | Comparative Study | lld:pubmed |
