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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
|
pubmed:dateCreated |
1979-11-21
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pubmed:abstractText |
Inflammatory stress as characterized by infection with Streptococcus pneumoniae, administration of endotoxin, or the induction of a turpentine abscess is characterized by the inhibition of the ketosis assoicated with fasting and a decline in the level of free fatty acids in the plasma. Moreover, rats subjectd to these inflammatory stresses demonstrate a significant rise in peripheral and portal insulin and glucagon. Rats subjected to noninflammatory stresses, screen-restraint, or noninvasive femoral fracture did not demonstrate the inhibition of ketosis but did show a decrease in plasma free fatty acids. The noninflammatroy stresses did not show an abnormal elevation of plasma or portal insulin or glucagon.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0360-3997
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
3
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
289-94
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:478598-Animals,
pubmed-meshheading:478598-Blood,
pubmed-meshheading:478598-Fatty Acids, Nonesterified,
pubmed-meshheading:478598-Glucagon,
pubmed-meshheading:478598-Inflammation,
pubmed-meshheading:478598-Insulin,
pubmed-meshheading:478598-Ketone Bodies,
pubmed-meshheading:478598-Male,
pubmed-meshheading:478598-Portal System,
pubmed-meshheading:478598-Rats,
pubmed-meshheading:478598-Restraint, Physical,
pubmed-meshheading:478598-Starvation,
pubmed-meshheading:478598-Stress, Physiological
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pubmed:year |
1979
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pubmed:articleTitle |
Effect of inflammatory and noninflammatory stress on plasma ketone bodies and free fatty acids and on glucagon and insulin in peripheral and portal blood.
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pubmed:publicationType |
Journal Article
|