Linked Life Data

465407

Source:http://linkedlifedata.com/resource/pubmed/id/465407

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
1979-10-17
pubmed:abstractText
This paper investigates the hypothesis that intermittent partial angle closure is one of the causes of ocular hypertension. 139 eyes from 76 patients with ocular hypertension were provoked with pilocarpine and phenylephrine. Four distinct responses appeared. Firstly, in 39 eyes (from 24 patients, 32%) gonioscopic closure of part or all of the angle appeared. Secondly, in 30 eyes (from 19 patients, 25%) no angle closure occurred but there was a substantial pigment release into the aqueous. Thirdly, in 9 eyes (from 8 patients, 11%) both angle closure and pigment release occurred. Finally, in 61 eyes (from 36 patients, 59%) neither angle closure nor pigment release appeared. (Since the 2 eyes of a patient did not always behave in the same way, for example, 1 eye might develop angle closure and the other not, 1 patient may appear in 2 groups.) From the first group 1 eye from each patient was randomly chosen for iridectomy. A repeat provocative test at least 1 year later produced a significantly different result. It is considered that the evidence obtained in this study supports the hypothesis that intermittent partial angle closure is one of the causes of ocular hypertension.
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0007-1161
pubmed:author
pubmed:issnType
Print
pubmed:volume
63
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
325-30
pubmed:dateRevised
2009-11-18
pubmed:meshHeading
pubmed:year
1979
pubmed:articleTitle
Mechanisms in ocular hypertension.
pubmed:publicationType
Journal Article, Clinical Trial, Randomized Controlled Trial