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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
|
pubmed:dateCreated |
1979-5-26
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pubmed:abstractText |
Bartter's syndrome characteristically exhibits the constellation of hypokalemic alkalosis with moderate kaliuresis, normotensive hyperreninemia, hyperaldosteronism, urinary hyperexcretion of prostaglandin E (PGE) and vascular hyporesponsivity to pressor agents. We describe precise biochemical mimicry of these metabolic abnormalities in a patient with surreptitious repetitive vomiting, in whom simple urinary chloride excretion data subsequently excluded the diagnosis of Bartter's syndrome.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0002-9343
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
66
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
361-3
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:425977-Adult,
pubmed-meshheading:425977-Bartter Syndrome,
pubmed-meshheading:425977-Chlorides,
pubmed-meshheading:425977-Diagnostic Errors,
pubmed-meshheading:425977-Humans,
pubmed-meshheading:425977-Hyperaldosteronism,
pubmed-meshheading:425977-Male,
pubmed-meshheading:425977-Potassium,
pubmed-meshheading:425977-Sodium Chloride,
pubmed-meshheading:425977-Vomiting
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pubmed:year |
1979
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pubmed:articleTitle |
Metabolic mimicry of Bartter's syndrome by covert vomiting: utility of urinary chloride determinations.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Case Reports
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